A team led by the University of Sydney has identified red blood cell rupture at dying endothelial sites as a primary driver of microvascular obstruction in COVID-19, bypassing the expected role of fibrin and platelet clots. Cases of severe injury to the body’s smallest blood vessels emerged during the COVID-19 pandemic, implicated in both sudden organ failure and persistent symptoms that span months. Tissue from affected patients reveals extensive endothelial damage across lung, heart, kidney and liver vasculature.
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