UNMC_Acronym_Vert_sm_4c
University of Nebraska Medical Center

Yulong Li, MD, PhD

Professor, UNMC Department of Emergency Medicine
Courtesy Professor, UNMC Department of Cellular & Integrative Physiology
Director of Basic Science and Translational Research, UNMC Department of Emergency Medicine

402-559-5413

Yulong Li, MD, PhD

Yulong Li, MD, PhD, is a professor of emergency medicine and a courtesy professor of cellular and integrative physiology. He is also a director of basic science and translational research in Department of Emergency Medicine. He is a research scientist th on that focuses on the regulatory role of the peripheral nervous system in peripheral tissues (such as myocardium and skeletal muscle) in pathophysiological conditions including heart failure, diabetes, ischemia-reperfusion injury and peripheral artery disease.

Before joining into the Department of Emergency Medicine, Dr. Li worked for five years in the Department of Cellular and Integrative Physiology at UNMC and his research interest was concentrated on neuronal control of cardiovascular system in the chronic heart failure. He also spent six years in Japan, where he would take some studies for the involvement of cardiac myocyte ion channels in cardiac arrhythmias and cardioprotection.

Education
  • MD: Hebei Medical University, Clinical Medicine, 1984.
  • Master's: Hebei Medical University, Cardiovascular Pharmacology, 1987.
  • PhD: Hebei Medical University, Cardiovascular Physiology, 1994.
Research Interests
  • Molecular and cellular signaling pathways.
  • Heart failure.
  • Diabetes.
  • Ischemia-reperfusion injury.
  • Peripheral artery disease.
  • Neuroscience.
Selected Publications
  • Red blood cell membrane-camouflaged poly(lactic-co-glycolic acid) microparticles as a potential controlled release drug delivery system for local stellate ganglion microinjection, 2023, Acta Biomaterialia.
  • Different responses of skeletal muscles to femoral artery ligation-induced ischemia identified in BABL/c and C57BL/6 mice, 2022, Frontiers in Physiology.
  • Macrophage depletion in stellate ganglia alleviates cardiac sympathetic overactivation and ventricular arrhythmogenesis by attenuating neuroinflammation in heart failure, 2021, Basic Research in Cardiology.