Discovery May Give Physicians More Tools for Predicting Patient’s Prognosis;


UNMC,  National Researchers Discover Common Cancer

is Two Distinct Diseases

For pathologists, distinguishing among the many types of B-cell lymphoma

has always been a challenge.  Current tumor classification systems

are based on subtle differences in the appearance of these immune cells

under a microscope, leaving many to wonder whether looks sometimes might

be deceiving.

But today in the weekly international journal, Nature, a team of scientists

report that in the future, there may be a new way around this old problem.

Using DNA microarray technology — a powerful new research tool that can

record the expression patterns of thousands of genes at once — the group

was able to show that, as currently defined, diffuse large B-cell lymphoma,

the most common form of non-Hodgkins lymphoma is actually two distinct

diseases.

Researchers at the University of Nebraska Medical Center, and collaborators

from five other institutions across the United States, say this information

may ultimately be used to predict the prognosis of patients with lymphoma.

Besides UNMC, other institutions involved in the research include: the

National Institutes of Health, Washington, D.C.; Stanford University School

of Medicine in Palo Alto, Calif.; Research Genetics in Huntsville, Ala;

Johns Hopkins School of Medicine in Baltimore; and Walter Reed Army Medical

Center in Washington, D.C.

Through this pilot project, we believe that genetic profiling will

be able to predict which lymphoma will have a good or poor prognosis for

the patient, said Wing (John) Chan, M.D., a pathologist and professor

in the UNMC Department of Pathology and Microbiology. Dr. Chan is principal

investigator of the grant at UNMC. One type of tumor may require aggressive

treatment while another may require less aggressive treatment, which would

reduce the potential complications involved with treatment. If we can find

out the genetic factors that determine prognosis and response to treatment,

then we may be able to use this information as a target to design new therapies

to attack the disease, Dr. Chan said.

Louis Staudt, M.D., Ph.D., a scientist at the National Cancer Institute

(NCI) and the senior author on the paper, said this finding helps to explain

why about 40 percent of patients with this type of NHL can be cured with

standard chemotherapy regimens, while other patients who seemingly have

the same disease often relapse.  Its a case of mistaken identity,

Dr. Staudt said.  The tumor cells might look very similar, but this

study offers strong


evidence that their molecular engines work very differently.

This weeks paper also adds to the growing interest among scientists

to create molecular profiles of common cancers.  This finding offers

one of the first glimpses into how cancer will be diagnosed in the future,

said NCI Director Richard Klausner, M.D. It will be based on well-characterized

biological differences among tumor cells that tell us more precisely how

aggressive a tumor will be and how best to treat it.

As the experiments proceeded, the scientists discovered something interesting.

The diffuse large B-cell lymphoma (DLBCL) samples showed distinct differences

in their expression of hundreds of genes, suggesting that DLBCL might be

more than one cancer.

An analysis showed two distinct patterns of gene expression, signifying

that they were looking at two subtypes of the cancer.  In one of the

subtypes, called GC B-like, the expression of germinal center genes was

largely congruent with that of normal B cells. But in the second subtype,

known as activated B-like, these genes were expressed at low or undetectable

levels. This finding that the subtypes express germinal center genes

differently–pointed to major biological differences among them.

In a pilot study of 42 previously untreated DLBCL patients who were

being treated with anthracycline-based chemotherapeutic regimens, the scientists

discovered significant differences in five-year survival rates among the

subtypes.  For patients with GC B-like, three-fourths were alive

at the five-year mark; for activated B-like, only 16 percent were still

living.

Ron Levy, M.D., an author on the paper and a scientist at the Stanford

University Medical Center, agrees. As this finding shows, genetic profiling

of tumors will offer extra power in predicting which patients will do well

and which will do poorly, he said. This more precise diagnosis of a developing

cancer should help in the future in more accurately guiding a patients

treatment decisions.

A five-year, $3.24 million grant was funded by the NIH at UNMC and collaborating

insititutions to verify and extend the groups finding, a standard practice

in science.  Diffuse large B-cell lymphoma is diagnosed in over 25,000

Americans each year.  At a time when many cancers are decreasing in

the United States, this lymphoma is increasing, making it a growing public

health concern.

The study also prompted a planned expansion of involvement from three

institutions in the United States and Canada to potentially eight in the

U.S., Canada and Europe. To confirm our initial finding, we need to have

a large number of cases to study. This would give us statistical power

to get a more firm conclusion, Dr. Chan said.

Four other UNMC researchers and clinicians also are involved in the

study. They are Timothy Greiner, M.D., Dennis Weisenburger, Ph.D., James

O. Armitage, M.D., and James Lynch, Ph.D.

UNMC is the only public academic health science center in the state.

Through its commitment to research, education, outreach and patient care,

UNMC has established itself as one of the country’s leading centers for

cancer research and treatment, solid organ transplantation and arthritis.

Nearly $32 million in research grants and contracts were awarded to UNMC

scientists during the past fiscal year. In addition, UNMCs educational

programs are responsible for training more health professionals practicing

in Nebraska than any other institution.


 

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